Smithsonian_03_2020

(Ann) #1
March 2020 | SMITHSONIAN 41

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breathing rate and the heart rate and expands all the blood
vessels, trying desperately to increase the amount of blood
being pumped past the lungs so they can process and elim-
inate the deadly gas. In the end stages of carbon dioxide ex-
posure, the acid in the veins begins to chemically break down
the myriad enzymes and proteins that control bodily func-
tions on a cellular level.
To do the math on the breathing gases, I would need the
boat’s interior volume, and to get that I would have to resort
to some scientifi c sleuthwork. Over the course of a month, I
downloaded every photo and diagram I could fi nd of the Hun-
ley and measured them meticulously to fi nd all the sub’s rel-
evant dimensions. After I used this information to create a
three-dimensional model, my computer could tell me the size.
Based on the interior volume of the boat, I calculated
how long it would take for painful levels of CO 2 to build up,
and determined the crew’s precise oxygen supply. The crew
would have had a 30- to 60-minute window of warning—de-
pending on their levels of physical exertion—between the
time the air fi rst reached a noticeable 5 percent CO 2 and
when it reached the low-oxygen level of 6.3 percent at which
they might pass out. Carbon di-
oxide causes pain; the headache
is sharp and profound, and the
ragged panting feels like the body
is struggling to catch up after a
panicky sprint. It was implausible
that the crew would have stayed
peaceful and quiet for this length
of time during such symptoms.
I had surpassed the threshold of
reasonable scientifi c evidence, and
therefore, for me, the theories of
suff ocation and asphyxiation were
eliminated. Once I ruled out those
theories, I turned back to examin-
ing my primary suspect: the blast.

THE AUTHOR KURT VONNEGUT once spoke in an interview
about his time in the military in Germany during World War
II, right after the fi rebombings that devastated Dresden. His
job had been to excavate the bomb shelters and basements
to remove the rotting corpses before the entire city started to
stink of human putrefaction. The people he found had usu-
ally died without moving, without any signs of struggle, and
were often still seated in their chairs. They were not outwardly
wounded; they were not blown wildly across the room.
There are multiple ways for victims to die in a fi rebombing,
and Vonnegut’s cases cannot be retroactively declared to have
all occurred solely because of one single cause. However, they
share the same key descriptors as the Hunley’s: undisturbed,
no external injuries, dead where they sat or stood. To a blast
researcher, this scenario sets off all the mental alarms. It starts
our heads screaming that we should at least suspect what is
called by our fi eld a “primary blast injury.”
Medically speaking, the injuries from an explosion are neatly
divided into one of four categories. A blast victim can receive
only one type of injury, or they can receive a grab bag of trauma
containing any mixture of the four. The injury types are num-

DISTANCE
FROM^ EXPLOSION

10 f

t

5 fe

et

20

ft
15
ft
30

ft

Hinged spar Torpedo

Trigger rope

Front hatch Captain Cross section
Bow

Snorkel

A PRIMARY


BLAST INJURY IS A


STRANGE AND


HORRIFYING FLUKE.

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