THE CURE FOR ALL ADVANCED CANCERS
show the presence of Sudan Black B dye. They are now finding
it and eating it. But to our dismay, it causes the same mutation
in them! Now their LDH levels go way up; it is spewed into the
blood stream now, where they are traveling, where it can affect
other organs and, of course, where it can be detected in the
blood test. Any organ that dares to pick up some of this LDH
will have its Krebs cycle suddenly curtailed since the pyruvate
is now being changed to lactic acid.
So you can expect the LDH levels to go up at first as immu-
nity improves! Symptoms are worsened. We must hurry. The
affected WBCs must be helped with selenite supplementation to
unload their toxic cargo in the kidneys and bladder for excre-
tion.
The second cause of LDH elevation is indirect. When cells
have lanthanide elements within them, a family of nucleoside
analogs appear called dideoxy nucleosides. How this happens is
not known, but that nucleoside analogs cause lactic acid eleva-
tion is well known.^117 It is the side-effect of drugs used for
AIDS patients. Raising lactic acid would inevitably raise LDH.
This needs further study.
By removing lanthanides with lightning speed (dental work
followed by magnet therapy) as well as excess ferritin, the LDH
drops without first rising and can be expected to drop 100 points
in just five days, sometimes faster.
Remember that a normal LDH doesn’t mean you are cancer
free. Not all cancer sufferers have an elevated LDH. But once it
begins to rise in a cancer sufferer, it spells doom because other
enzymes are mutated too; all body tissues will soon be dysfunc-
tional. Hurry to get the dyes (dental plastic, etc.) out of your
body! And avoid eating both dyes and lanthanides, as discussed
in Food Rules.
When the LDH is very high, over five hundred, all the dye
treatments should be maximized. Normally, we reduce dyes
with coenzyme Q10 and vitamin B 2. But if the LDH is over one
thousand and all your treatments are not bringing it down in the
(^117) Positively Aware, Jan/Feb 1999, p.31.