Aortic Injury/Traumatic Rupture of the Aorta (TRA)
MECHANISMS
■ Consider in the setting of high-speed deceleration and lateral impact MVCs
■ Consider in presence of multiple rib fractures or flail chest (although one-
third of blunt aortic injuries have no obvious external thoracic injury)
SYMPTOMS/EXAM
■ Retrosternal or interscapular pain
■ Dysphagia
■ Shortness of breath
■ Stridor or hoarseness in the absence of laryngeal injury
■ Harsh murmur over precordium or space between the scapula
■ Signs of superior vena cava syndrome
■ Hypotension
DIAGNOSIS
■ Compare BP in upper versus lower extremities (upper extremity hypertension
suggests acute coarctation syndrome).
■ CXR (10% of initial CXR in patients with TRA are completely normal)
(see Table 3.8)
■ Widened mediastinum(see Figure 3.12)
■ Defined by a width >8 cm on a supine AP film
■ Sensitivities are estimated at 50–92%.
■ Specificity is estimated at 10%. Often the mediastinum will be wide
in patients who do not have TRA.
■ Esophageal deviation
■ Loss of distinct aortic knob
■ Loss of paraspinal stripe/displacement of right paraspinous interface
■ Widening of the right paratracheal stripe
■ Loss of clear space between aortic knob and left pulmonary artery
(apical cap)
■ Displacement of left main stem bronchus 40° below horizontal TRAUMA
TABLE 3.8.CXR Findings Suggesting Aortic Injury
Widened mediastinum
Esophageal deviation
Loss of distinct aortic knob
Loss of paraspinal stripe/displacement of right paraspinous interface
Widening of the right paratracheal stripe
Loss of clear space between aortic knob and left pulmonary artery (apical cap)
Displacement of left main stem bronchus 40° below horizontal
Dissection of the descending
aorta (Stanford type B or
Debakey type III) is usually
managed medically.
Traumatic rupture of the aorta
is almost always treated
surgically, regardless of
location.
Half of all patients with TRA
who reach the hospital and
survive for 1 hour die within
24 hours, and 75% die within
7 days.