CRANIAL NERVE DISORDERS
Trigeminal Neuralgia
The trigeminal nerve (CN V) has three anatomic divisions, V 1 -ophthalmic, V 2 -
maxillary, and V 3 -mandibular, which innervate the cornea, the face, and the
mucous membranes of the oral and nasal cavity. The motor fibers innervate
the muscles of mastication.
SYMPTOMS/EXAM
■ Brief, recurrent episodes of excruciating, unilateral facial pain
■ Right > left side predominance
■ May be able to elicit pain by tapping the side of the face, otherwise there
should be no demonstrable physical findings
DIFFERENTIAL
■ Includes vascular or space-occupying lesions (acoustic neuroma), demyeli-
nating diseases (multiple sclerosis), herpes zoster, sinus infection, odonto-
genic pathology, migraine, temporomandibular joint dysfunction
DIAGNOSIS
■ Based on clinical presentation
■ Imaging should be performed to rule out other etiologies if neurological
findings are present.
NEUROLOGY
TABLE 15.12. Disorders Causing Peripheral Vertigo
DISORDER PATHOPHYSIOLOGY ASSOCIATED FINDINGS
Benign paroxysmal positional Otoconia in the semicircular canals Precipitated by sudden head movement
vertigo (BPPV) Positive Dix-Hallpike
Improvement/resolution with Epley maneuver
Meniere disease Increased endolymph within the Ear “fullness,” tinnitus, hearing loss
cochlea and labyrinth
Labyrinthitis Viral or bacterialinfection Middle-ear findings (infection, fluid), tinnitus,
hearing loss
Vestibular neuronitis Viral infection Lasts several days, no recurrence or hearing loss
Ramsay-Hunt syndrome Viral infection of vestibular gangion Hearing loss, vertigo, facial nerve palsy, grouped
(vestibular ganglionitis) vesicles
Perilymph fistula Trauma, sudden pressure change Abrupt onset after inciting event +/−hearing loss
Requires surgical repair
Ototoxicity Damage to vestibular apparatus Hearing loss, vertigo, tinnitus
—may be irreversible
CN VIII lesions Schwannomas, meningioma Gradual onset, preceded by hearing loss
Treatment for trigeminal
neuralgia = Tegretol
(carbamazepine)