The China Study by Thomas Campbell

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  1. Mainigi KD, and Campbell Te. "Effects of low dietary protein and dietary aflatoxin on he-
    patic glutathione levels in F-344 rats." Toxicol. Appl. Pharmacol. 59 (1981): 196-203.

  2. Farber E, and Cameron R. "The sequential analysis of cancer development." Adv. Cancer Res.
    31 (1980): 125-226.

  3. Foci response for the various charts in this chapter mostly reflect "% of liver volume," which
    integrates "number of foci" and "size of foci," both of which indicate tumor-forming ten-
    dency. So that the responses from individual experiments can be compared among each other,
    the data are adjusted to a common scale that reflects the response produced by a standard
    dose of aflatoxin and by feeding a 20% protein diet.

  4. Appleton BS, and Campbell Te. "Inhibition of aflatoxin-initiated preneoplastic liver lesions
    by low dietary protein." Nutr. Cancer 3 (1982): 200-206.

  5. Dunaif GE, and Campbell Te. "Relative contribution of dietary protein level and Aflatoxin
    B) dose in generation of presumptive preneoplastic foci in rat liver." ]. Natl. Cancer Ins1. 78
    (1987): 365-369.

  6. Youngman LD, and Campbell Te. "High protein intake promotes the growth of preneoplastic
    foci in Fischer #344 rats: evidence that early remodeled foci retain the potential for future
    growth."]' Nulr.121 (1991): 1454-1461.

  7. Youngman LD, and Campbell Te. "Inhibition of aflatoxin Bl-induced gamrna-glutamyl
    transpeptidase positive (GGT+) hepatic preneoplastic foci and tumors by low protein diets:
    evidence that altered GGT+ foci indicate neoplastic potential." Carcinogenesis 13 (1992):

  8. Dunaif GE, and Campbell Te. "Dietary protein level and aflatoxin Bl-induced preneoplastic
    hepatic lesions in the rat." ]. Nutr. 117 (1987): 1298-1302.

  9. Horio F, Youngman LD, Bell RC, et al. "Thermogenesis, low-protein diets, and decreased de-
    velopment of AFBl-induced preneoplastic foci in rat liver." Nutr. Cancer 16 (1991): 31-41.

  10. About 12% dietary protein is required to maximize growth rate, according to the National
    Research Council of the National Academy of Sciences.

  11. Subcommittee on Laboratory Animal Nutrition. Nutrient requirements of laboratory animals.
    Second revised edition, number 10. Washington, DC: National Academy Press, 1972.

  12. National Research Council. Recommended dietary allowances. Tenth edition. Washington, DC:
    National Academy Press, 1989.

  13. Schulsinger DA, Root MM, and Campbell Te. "Effect of dietary protein quality on develop-
    ment of aflatoxin Bl-induced hepatic preneoplastic lesions."]' Natl. Cancer Inst. 81 (1989):

  14. Youngman LD. The growth and development of ajlatoxin Bl-induced preneoplastic lesions, tu-
    mors, metastasis, and spontaneous tumors as they are influenced by dietary protein level, type, and
    intervention. Ithaca, NY: Cornell University, Ph.D. Thesis, 1990.

  15. Beasley RP. "Hepatitis B virus as the etiologic agent in hepatocellular carcinoma-epidemio-
    logic considerations." Hepatol. 2 (1982): 215-265.

  16. Blumberg BS, Larouze B, London WT, et al. "The relation of infection with the hepatitis B
    agent to primary hepatic carcinoma." Am.]. Pathol. 81 (1975): 669-682.

  17. Chisari FY, Ferrari C, and Mondelli MU. " Hepatitis B virus structure and biology." Microbiol.
    Pathol. 6 (1989): 311-325.

  18. Hu j, Cheng Z, Chisari FY, et al. "Repression of hepatitis B virus (HBV) transgene and HBV-
    induced liver injury by low protein diet." Oncogene 15 (1997): 2795-2801.

  19. Cheng Z, Huj, Kingj, et al. "Inhibition of hepatocellular carcinoma development in hepatitis
    B virus transfected mice by low dietary casein." Hepatology 26 (1997): 1351-1354.

  20. Hawrylewicz Ej, Huang HH, KissanejQ, et al. "Enhancement of the 7,12-dimethylbenz(a)a
    nthracene (DMBA) mammary tumorigenesis by high dietary protein in rats." Nutr. Reps. Int.
    26 (1982): 793-806.

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