24 Acidic Wastes: The Real Culprit
cals in the gastrointestinal tract. But while the meat eater–grain eater
plan normalizes stomach acid levels—important because acid initiates
the breakdown of protein—Theron Randolph’s four-day rotation diet
(see Randolph’s book in the Bibliography) normalizes the protein-
digesting enzymes, which complete the digestion of protein.
Randolph’s four-day rotation diet works well in terms of increasing
food-digesting enzyme levels, because, with no single food eaten more
than once every fi ve days, digestive enzymes involved in the breakdown
of protein, carbohydrates, and fats have a chance to build up. In the
four-day rotation diet even fats and oils are rotated. For example, butter
on day one, olive oil on day two, the third day coconut oil, and on the
fourth, grapeseed oil. On the fi fth day the cycle starts all over again. You
go back to butter as well as all the other food items you ate on day one.
Before you start the four-day rotation diet, give up caffeine, alcohol,
and cigarettes, and for the fi rst three months avoid those foods you eat
three or more times a week, since it’s the foods eaten most frequently
that are likely to cause allergies. Dr. William Philpott, a neurologist
who specialized in magnets, was so enthusiastic about the four-day
rotation diet that when people called him to order magnets, before he
took their order he urged them to go on this diet. Philpott himself was
one of its benefi ciaries. According to his wife he was healthier at age
eighty-nine than he was at fi fty—before he went on the rotation diet
that cured him of type 2 diabetes and arthritis.
Randolph’s diet is especially effective in reversing type 2 diabetes,
which nowadays strikes young adults as well as the middle-aged, prob-
ably because by the age of thirty most people are defi cient in enzymes.
When type 2 diabetics go on Randolph’s diet, they increase their
enzyme reserves. This helps in the disposal of blood sugar that cannot
infi ltrate the cells, a problem endemic in type 2 diabetics. Although the
beta cells in the pancreas continue to secrete insulin, in type 2 diabetes
there is an insulin-resistance problem. I suspect it is not insulin that
prevents the cell’s absorption of glucose, but the glucose transport car-
riers within the cells. In type 2 diabetes, these carriers cannot move
from the interior of the cell to the cell membrane to help the glucose
molecules pass through the cell’s receptors (entry points).
Thus glucose’s transportation into the cells depends not only on
insulin but also on the transportation system inside the cell. When kept
from entering the cells, glucose accumulates in the blood. The four-day