Sports Medicine: Just the Facts

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CHAPTER 26 • DERMATOLOGY 153

turn grayish-white, develop paresthesias, and finally
lose sensation.


  • Penile frostnip has been reported in joggers wearing
    polyester trousers and cotton undershorts (Hershkowitz,
    1977).

  • Frostnip can be reversed with immediate self-rewarm-
    ing of the exposed area.
    •Paresthesias and a burning sensation may persist for
    several months after the injury.

  • Prevention includes insulation and skin protection
    from both wind and cold, not shaving prior to partici-
    pation to preserve the natural skin oils and application
    of a sunscreen. (D’Ambrosia, 1977)


FROSTBITE



  • Frostbite occurs as living tissue ceases cellular metab-
    olism from exposure to temperatures below 28°F
    (–2°C)(D’Ambrosia, 1977).

  • Muscles, nerves and blood vessels are damaged ear-
    lier than tendons and bone (D’Ambrosia, 1977).

  • The tissue appears cold, white, and hard and will not
    exhibit pain or sensation to tactile stimulation until
    thawing occurs.
    •Rewarming of the tissue should be attempted only
    when the environment can be controlled and risk of
    refreezing of the affected part is eliminated.

  • Frozen areas should be rewarmed as rapidly as possible
    in a warm-water, circulating bath of 110–112°F (38–
    44 °C) to prevent mechanical trauma (D’Ambrosia,
    1977).
    •Tissue necrosis may occur for weeks to months as
    reepithelialization replaces the denuded areas (Levine,
    1980).


COLDURTICARIA



  • Cold urticaria is rare but the most common form of
    acquired physical urticaria in winter athletics and cold
    water swimming (Mikhailov, Berova, and Andreev,
    1977).

  • Wheals or hives are usually confined to the exposed
    area.

  • Athletes with cold urticaria are likely to have recurrent,
    severe episodes during similar circumstances because
    of the antigen–antibody reaction resulting in the
    release of histamine (D’Ambrosia, 1977).
    •To confirm the diagnosis, provocative testing of the
    forearm with ice cubes for 5 min or submerging the
    forearm in cold water for 5–15 min will produce
    wheals (Pharis, Teller, and Wolf, Jr, 1997).

  • The prophylactic use of cyproheptadine (Periactin), 2 mg
    once or twice a day, or doxepin (Sinequan), 10 mg two
    or three times daily have been useful in preventing
    reoccurrence (Williams and Batts, 2001).


INFECTIOUS INJURY

BACTERIAL

FURUNCULOSIS


  • Erythematous, nodular abscesses found in the hairy
    areas of the axilla, buttocks, and groin.
    •Highly contagious and known outbreaks have occur-
    red in team sports, implicating close contact and poor
    hygiene practices (Sosin et al, 1989).

  • Staphyloccocci are the most common bacteria and the
    nares should be cultured because of the predisposition
    of the nares to harbor the staphylococcus species
    (Adams, 2002a).

  • Acute treatment consists of warm compresses and a
    10-day course of a cephalosporin, erythromycin, or
    penicillinase-resistant penicillin derivative (Bergfeld
    and Helm, 1991).
    •A prophylactic dose of rifampin, 600 mg for 10 days,
    has been utilized in resistant cases (Williams and
    Batts, 2001).

  • Incision and drainage is necessary because of the poor
    hematogenous antibiotic penetration.

  • The NCAA requires all wrestlers to be without new
    lesions for 48 h before a meet, have completed 72 h of
    antibiotic therapy, and have no moist or draining
    lesions prior to competition (Bubb, 2002).


PITTEDKERATOLYSIS
•A scalloped-bordered plaque with sculpted pits of vari-
able depth forms on the weight-bearing plantar surfaces
(heel and toes) and is often misdiagnosed as tinea pedis.


  • Hyperhidrosis and gram-positive bacteria, most com-
    monly Corynebacterium and Micrococcus species,
    found in the stratum corneum have been implicated in
    producing the pungent foot odor (Schissel, Aydelotte,
    and Keller, 1999).

  • Application of topical antibiotics for 2–4 weeks (5%
    erythromycin in 10% benzoyl peroxide) will reduce
    the bacterial inflammatory component and result in
    clearing (Kantor and Bergfeld, 1988).

  • Prophylactic therapy includes washing with benzoyl
    peroxide soap and adding topical foot powders with
    20% aluminum chloride (Drysol) to control hyper-
    hidrosis (Schissel, Aydelotte, and Keller, 1999).


IMPETIGO


  • Serosanguinous, honey-crusted pustules on an erythe-
    matous base.

  • Beta-hemolytic streptococci more commonly produce
    impetigo, but staphylococcal species have been iso-
    lated from cultured wounds.

  • Athletes participating in contact sports or swimming
    are highly contagious and should not participate in

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