Sports Medicine: Just the Facts

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CHAPTER 45 • ROTATOR CUFF PATHOLOGY 269


  • Posterosuperior glenoid impingement: This condi-
    tion, as described by Walch (Walch et al, 1992), has
    also been proposed as an etiology in occurring in
    patients who play repetitive overhead sports such as
    baseball, tennis, and swimming. Walch did not find
    the anterior instability described by Jobe in his
    patients, but rather noted an impingement of the
    supraspinatus and infraspinatus tendons between the
    posterosuperior glenoid labrum and the humeral head.

  • These internal impingement syndromes are character-
    ized by partial tears of the articular surface of the rota-
    tor cuff, in distinction to the external compression
    described by Neer (McFarland et al, 1999).
    •Whatever the etiology, weakness of the rotator cuff
    results, especially the lower cuff, and superior humeral
    head migration occurs. The humeral head then com-
    presses the bursa and tendon into the acromion, lead-
    ing to impingement. This causes more bursitis, more
    tendinosis, and eventually more weakness. Often with
    chronic injury, shoulder mechanics will change lead-
    ing to abnormal scapulothoracic motion. Physical ther-
    apy will need to include rehabilitation of the scapular
    stabilizing musculature as well as the lower rotator
    cuff muscles (Jobe and Moynes, 1982).
    •Any subacromial changes, such as lateral hooking,
    CA ligament calcification, or AC joint arthritis (infe-
    rior spurs), will cause the condition to get worse and
    will more likely need operative intervention than
    when the acromion is flat.

  • Calcific tendinitis: The etiology of calcific tendinitis
    remains unknown. Degenerative changes and relative
    hypoxia has been suggested as possible explanations.
    Conservative treatment similar to that for rotator cuff
    syndrome is reported as successful in 60–90% of
    patients (DePalma and Kruper, 1961; Harmon, 1958;
    Moseley, 1963). Repetitive needling and injection of
    local anesthetic has also been successful in relieving
    symptoms and often disappearance of the calcified
    mass. Infrequently, the patient’s symptoms will per-
    sist and they will require operative intervention. The
    mass is localized within the substance of the tendon
    while viewing in the subacromial space. The calcified
    substance is then evacuated and debrided. Some argue
    that a repair of the tendon is not necessary, but the
    surgeon should evaluate the cuff after debridement in
    each case to determine if repair is necessary.


EXAMINATION (YAMAGUCHI
ET AL, 2001)



  • Inspection should focus on normal alignment of chest
    wall, shoulders, and clavicle. Have the patient perform
    active range of motion (ROM) in forward flexion,


abduction, adduction, external rotation, and internal
rotation. Look from the back and front for asymmetric
motion or atrophy. Often patients will have a painful
arc of motion over 120°of elevation.
•Palpation of the AC and sternoclavicular(SC) joints,
the anterior and lateral acromion edges and the infra-
spinatus fossa. Tenderness at the AC joint should lead
you to further evaluation and treatment of AC joint
arthrosis. Cross-arm adduction is often painful with
AC arthrosis; however, this test is not very specific
and is often positive with rotator cuff syndrome.
•Palpation of the long head of the biceps tendon within
the bicipital groove is helpful to determine biceps
involvement.


  • Strength testing should involve the deltoid muscles,
    biceps muscles, and triceps muscles. Although there is
    no way to totally isolate each of the rotator cuff mus-
    cles, the tests that have shown to be most specific are
    as follows (Kelly et al, 1996):

    1. Supraspinatus: Active elevation against resistance
      with the elbow in extension and the arm elevated to
      90 °and externally rotated to 45°. The hand should be
      supinated to neutral as if holding a full can of soda.

    2. Infraspinatus/teres minor: Active external rotation
      with arm at the side and elbow flexed to 90°.

    3. Subscapularis: Active internal rotation with elbow
      flexed to 90°and hand placed behind the back. This
      is often referred to as the Gerber lift-off test (Gerber,
      Hersche, and Farron, 1996; Gerber and Krushell,
      1991; Greis et al, 1996). In patients who are unable
      to internally rotate their hand behind their back, a
      belly press test or “Napoleon’s test” is performed.
      Patient place their hands on their belly and press
      hard into their abdomen while bringing their elbow
      forward in the sagittal plane. Subscapularis tear or
      dysfunction is indicated if they are unable to do this
      maneuver and the elbow stays close to the side
      (Warner, Allen, and Gerber, 1994).



  • Lag tests are also often used to detect rotator cuff
    tears. The Hornblower’s sign, ER lag and IR lag tests
    were described by Gerber and Hertel and are helpful
    to determine subtle weakness (Gerber and Krushell,
    1991; Hertel et al, 1996).

  • Special tests include the Neer impingement sign and
    test and Hawkins’ sign.
    1.Neer’s impingement sign is similar to the
    supraspinatus testing described above, except the
    arm is held in maximal internal rotation as if pour-
    ing out a can of soda. This rotates the greater
    tuberosity under the acromion to elicit a painful
    response if the bursa or tendons are injured. A pos-
    itive Neer’s test is when a subacromial injection of
    local anesthetic relieves the pain elicited prior to
    the injection (Neer, 1972; 1983).

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