b.Repair is considered in those lesions greater
than 50% of tendon thickness (normal 12–
16 mm).
c. Debridement is in lower grade lesions and in
those with normal preoperative strength.
d. Consider acromioplasty and/or coracoacromial
ligament release.
IMPINGEMENT
PRIMARY
- Rare as an isolated entity.
- High incidence of intra-articular pathology.
- Arthroscopy indicated in all patients to assess for
additional pathology. - Conservative resection.
a. Do not resect type I acromion.
b. Release of coracoacromial ligament (no resection).
SECONDARY
- Most often missed and inappropriately treated as pri-
mary problem:- Requires treatment of the primary problem either
with rehabilitation or surgical intervention.
- Requires treatment of the primary problem either
- Arthroscopy helps assess for primary intra-articular
pathology. - The employment of subacromial decompression in
isolation has been shown to be unpredictable with
respect to return to prior activity level.
a. In one study, only 43% of patients with surgical
decompression returned to preinjury level of com-
petition (Tibone et al, 1985).
b. Return is perhaps more indicative of improvement
in their muscular imbalance through thoughtful
rehabilitation than as a result of the surgical proce-
dure.
REHABILITATION ISSUES
SCAPULOTHORACICARTICULATION
- Often the cause of secondary impingement.
- Scapula has five specific functions that have implica-
tions in throwers (Kibler, 1998).
a. Stable part of the glenohumeral articulation, where
rotation of the glenohumeral joint allows maximal
concavity and compression.
b. The scapula retracts and protracts the shoulder
complex along the thoracic wall.
c. Acromial elevation to avoid impingement with arm
elevation.
d. Base for muscular attachments.
e. Energy transfer from the legs, back, and trunk.
SCAPULOTHORACICDYSKINESIA
- Abnormal set of motions and positions affecting the
relative position of the scapula and the proximal
humerus.
a. Etiologies include nerve or muscle injury, muscle
inhibition, and glenohumeral stiffness or laxity.
b. Mechanical dysfunction may result in impinge-
ment as well as insufficient translation of energy
from the lower body.- Excessive stress results in overuse injuries.
- Clinical picture is confusing as a result of sec-
ondary impingement and capsular changes that
may occur as a result of the adaptations to
scapular malalignment.
PROPRIOCEPTION(LEPHART ANDHENRY, 2000)
- Excessive joint laxity associated with capsuloliga-
mentous injury and resulting microtrauma cause
damage to the neural receptors and lead to deaf-
ferentation. - Neuromuscular deficits impair reflexive muscular sta-
bilization, predisposing shoulder to episodes of func-
tional instability.
a. Diminished joint position sense, kinesthetic aware-
ness, and abnormal humeroscapular firing patterns
(Lephart et al, 1996).
b. Abnormal firing patterns documented on EMG
studies in throwers with glenohumeral instability
(Glousman et al, 1988).
c. Following surgical reconstruction, joint position
sense, and reproduction of passive positioning
improve to baseline levels (Lephart et al, 1996). - Restoration of functional stability.
a. Traditional strengthening exercises do not address
neuromuscular deficits.
b. Four elements necessary to restore functional sta-
bility (Lephart et al, 1996):- Peripheral somatosensory, including visual and
vestibular - Spinal reflexes:
- Sudden alteration in joint position that
requires reflex muscular stabilization.
- Sudden alteration in joint position that
- Cognitive programming:
- Appreciation of joint position.
- Brain stem.
c. All four elements need to be addressed in order
to fulfill the objective of stimulating all sub-
systems: - Dynamic stabilization:
a. Promotes coactivation of force couples
b. Centers humeral head - Joint position sensibility:
- Restore through conscious and unconscious
pathways.
- Restore through conscious and unconscious
- Peripheral somatosensory, including visual and
290 SECTION 4 • MUSCULOSKELETAL PROBLEMS IN THE ATHLETE