CHAPTER 88 • RUNNING 523
- Diagnosis
- Lateral pain and crepitus at lateral femoral condyle
(or insertion at Gerdy’s tubercle) - Tight ITB on OBER test
- Abductor weakness
- Lateral pain and crepitus at lateral femoral condyle
- Contributing factors
- Varus positioning of knee, tibia, and foot
- Internal tibial torsion
- Excessive supination or pronation
- Tightness abductors, adductors, ext rotators, and
hip extensors - Abductor weakness
•Treatment - PRICEMM (iontophoresis)
- Dual action strap
- Foam roller exercise for soft tissue work
- Flexibility and strength (hip abductors, adductors,
rotators, flexors, and extensors)
- Consider with persistent symptoms
- Injection (steroid)
- MRI r/o lateral meniscus and other
SHIN SPLINTS
- Definition
- Shin splints, or medial tibial stress syndrome, is a
clinical entity characterized by diffuse tenderness
over the posteromedial aspect of the distal third of
the tibia. Shin splints have been reported to
account for 12–18% of running injuries (James,
Bates, and Ostering, 1978; Briner, Jr, 1988; Gudas,
1980; Pinshaw, Atlas, and Noakes, 1984) and in
4% of all military recruits in basic training
(Andrish, Bergfeld, and Walheim, 1974). Women
appear more frequently affected than men. - Medial tibial stress syndrome is to be differentiated
from stress fractures and exertional compartment
syndrome. Although different entities, they may
coexist. Plain films are negative (except in cases of
previous or coexistent stress fracture). Bone scans
will demonstrate characteristic vertical linear
increased activity along the tibial periosteum,
which differs from the more focal fusiform
increased radiotracer uptake exhibited by stress
fractures. - Medial tibial stress syndrome is felt by most to
represent a periostalgia or tendinopathy along the
tibial attachment of the tibialis posterior or soleus
muscles. Other proposed etiologies have included
posterior compartment syndrome and fascial
inflammation. Detmer proposed a classification
scheme for medial tibial stress syndrome based on
etiology. Type 1 included local stress fractures,
- Shin splints, or medial tibial stress syndrome, is a
type 2 periostitis/periostalgia, and type 3 due to
deep posterior compartment syndrome (Detmer,
1986).
- Diagnosis
- Dull ache medial shaft with activity
- Tenderness to palpation along shaft
- Normal neurovascular examination and X-ray
- Contributing factors
- Factors that increase valgus forces and pronation,
which then increase eccentric contraction of the
soleus and tibialis posterior: femoral anteversion,
genu varum, tibia and forefoot varus, and excessive
Q angle - Excessive pes planus or cavus
- Tarsal coalation
- Leg length inequality
- Muscle imbalances: Inflexibility of plantar flexors;
weakness of dorsiflexors, plantarflexors, and
inventors - Extrinsic risk factors include improper shoe wear,
a rapid transition in training, inadequate warm-up,
running on uneven or hard surfaces, running in
cold weather, and low calcium intake.
•Treatment - Flexibility: Gastroc-soleus, tibialis posterior
- Strength: Concentric and eccentric including tibialis
posterior soleus, tibialis anterior, FH, FDL - Orthotic to control compensatory pronation
- Shin sleeve
- Factors that increase valgus forces and pronation,
- Consider with persistent symptoms
- Bone scan/MRI r/o stress fx
- Compartment testing to r/o compartment syndrome
- Consider lumbar radiculopathy
EXERTIONAL COMPARTMENT SYNDROME
- Definition
1.Chronic exertional compartment syndrome is
defined as reversible ischemia secondary to a non-
compliant osseofascial compartment that is unre-
sponsive to the expansion of muscle volume that
occurs with exercise. Most commonly seen in the
lower leg, exertional compartment syndrome in ath-
letes has also been described in the thigh and medial
compartment of the foot (Raether and Lutter, 1982;
Birnbaum, 1983; Mollica and Duyshart, 2002).
2. There are four major compartments in the leg.
Each is bound by bone and fascia, and each con-
tains a major nerve.
a. The anterior compartment contains the extensor
hallucis longus, extensor digitorum longus, per-
oneus tertius, and anterior tibialis muscles, as
well as the deep peroneal nerve.