Sports Medicine: Just the Facts

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CHAPTER 88 • RUNNING 525


  • Diagnosis

    1. Tenderness at myotendinous junction or insertion



  • Contributing Factors

    1. Pronation

    2. Lower extremity varus

    3. Tight heel cord

    4. Weak dorsiflexors/plantar flexors(DF/PDF)
      •Treatment

    5. Heel lifts

    6. Control pronation

    7. Flexibility (include gastroc and soleus)

    8. Strength (particulary eccentric strength)

    9. Modalities (ionto and cross-friction massage)



  • Consider if persistent

    1. Retrocalcaneal bursitis

    2. MRI (evaluate tears)

    3. Immobilization

    4. Lidocaine injections for periotendonitis




PLANTAR FASCIITIS



  • Definition

    1. An overload injury including inflammation degen-
      eration, and tearing of the plantar fascia, most
      commonly at its calcaneal insertion.



  • Contributing factors

    1. Tight gastroc soleus, plantar fascia

    2. Rigid rear foot

    3. Over pronation or supination



  • Diagnosis

    1. Pain plantar heel worse in a.m. and with activity

    2. Tenderness plantar medial heel

    3. Normal neuro examination

    4. No bony (calcaneal) tenderness
      •Treatment

    5. Phase 1:
      a.anti-inflammatories: Nonsteroidal anti-inflamma-
      torydrugs(NSAIDs) and medrol
      b.Device (CTF brace)
      c. Stretch (gastroc-soleus, plantar fascia, hamstring,
      ITB)
      d. Strength (foot intrinsics)

    6. Phase 2:
      a. P.T. (phono/ionto, massage)
      b.Continue exercise
      c. Different device (heel cushion)

    7. Phase 3: Night splint

    8. Phase 4,5: Injections (3 max), orthotics



  • Consider if persistent

    1. MRI: r/o calcaneal stress injury

    2. EMG/NCS: r/o Medial calcaneal, Plantar neuropa-
      thy, and radiculopathy

    3. Surgery if fail conservative care (6 months)




STRESS FRACTURES


  • Definition

    1. Failure of bone to adapt adequately to mechanical
      loads (ground reaction forces and muscle contrac-
      tion) experienced during physical activity.

    2. Tibial stress fractures predominate in distance run-
      ners. Navicular stress fractures predominate in
      track athletes.



  • Diagnosis

    1. Focal tenderness

    2. Recent transition in training

    3. X-ray (often neg., esp. early)

    4. Bone scan (all three phases with increased uptake)

    5. MRI (periosteal and marrow edema T 2 > T 1 )



  • Contributing factors

    1. Rapid transition in training

    2. Osteopenia/Osteoporosis

    3. Menstrual irregularities, in particular amennorrhea

      6 months.




    4. Excessive pes cavus (femoral, tibial) or pes planus
      (metatarsals)

    5. Poor shoe wear
      •Treatment
      1.Noncritical: Noncritical stress fractures can be
      treated with 6–8 weeks of relative rest and
      include the following: medial tibia, metatarsals 2,
      3, 4, and 5 metatarsal avulsion. Athletes may ben-
      efit from a short period (i.e., 3 weeks) in a walking
      boot.

    6. Critical stress fractures require more specific atten-
      tion due to slower healing and higher rates of
      nonunion and include the following: femoral neck,
      anterior tibia, medal malleolus, navicular, and base
      5th metatarsal.
      a. Femoral neck
      i. Normal X-ray, no cortical break: Conservative
      RX, partial weight-bearing(PWB) to weight-
      bearing as tolerated(WBAT). Follow clini-
      cally and serial MRI 8–12 weeks.
      ii. Cortical break: ortho. Superior (distraction)
      fractures have a higher incidence of worsen-
      ing and nonunion than inferior (compression)
      fractures.
      b.Anterior tibia
      i. Casting versus relative rest up to 6–8 months.
      ii. If no healing: Ortho (transverse drilling, graft-
      ing, medullary fixation)
      c. Medial malleolus
      i. Nondisplaced: Boot or aircast 6 weeks
      ii. Displaced or nonunion: Ortho
      d. Navicular
      i. Nonweight bearing 6–8 weeks
      ii. Progressive activity over 6 more weeks



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