Sports Medicine: Just the Facts

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on grass, where males had six times higher total
injuries and female injuries were 4.3 times higher
(Soderman, 2001).

SOCCER INJURY CHARACTERISTICS


ANKLE INJURY



  • The mechanism of an ankle sprain is often an inver-
    sion stress as the talus tilts in plantar flexion to
    manage tasks such as pivoting, jumping, and hard
    turns with the ball. In this position the anterior talo-
    fibular ligament(ATFL) is at maximal tension and
    most vulnerable to injury. The calcaneal fibular liga-
    ment(CFL) and posterior talo-fibular ligament follow
    in order of injury behind the ATFL. The spectrum of
    severity ranges from a lateral ankle ligament stretch
    (Grade 1) to partial tear (Grade 2) to complete tear
    (Grade 3).

  • Medial ankle ligament injuries are much more rare
    due to the dominant strength of the deltoid ligament.
    Therefore, a medial ankle disruption suggests a high-
    energy impact and will possibly demonstrate ankle
    mortise alteration requiring surgical management for
    fractures and follow a longer recovery period.

  • Soccer players are susceptible to high ankle injuries.
    During collisions or contested balls, the ankle position
    during impact can be in the talar neutral position, with
    or without a slight lateral talar tilt. This predisposes
    the athlete to greater stresses along the calcaneal fibu-
    lar ligament and the inferior anterior tibial-fibular lig-
    ament. Performing a proximal tib-fib “squeeze test”
    with distal referred pain at the ankle or instability with
    external foot rotation is suggestive for a high ankle
    sprain.

  • In the ankle with persistent pain and swelling beyond
    two weeks the differential diagnosis should include
    talar dome lesions, such as osteochondral fractures or
    a fracture to the lateral process of the talus. If clinical
    suspicion is high an MRI should be done.

  • The injured soccer player with chronic anterior ankle
    pain extending on to the midfoot who had an acute
    axial loading of the fore- and midfoot (kick into the
    turf) should be evaluated for a bifurcate ligament
    injury, impingement syndrome (especially for repeat
    ankle injuries) or Lisfranc fracture. If clinical suspi-
    cion dictates and weight bearing X-rays of the
    ankle/foot are normal then a MRI of the ankle/foot
    should be done.

  • Posterior ankle pain or hind foot pain in a soccer ath-
    lete should raise concern in the young athlete
    (Engstrom, Johanoson, and Tornkvist, 1991; Schmidt-
    Olsen et al, 1991; Albert, 1983; Nilsson and Roaas,


1978; National Collegiate Athletic Association, 2000;
Ekstrand and Gillquist, 1983) for Sever’s disease or
more commonly retrocalcaneal bursitis with indoor
soccer players who have a pattern of repetitive heel
trauma against the sideboards. Achilles tendinitis dif-
fers from the above two conditions by presenting sev-
eral centimeters proximally to the tendon insertion
into the calcaneus.


  • Chronic posterior ankle pain should draw concern for
    a calcaneal stress fracture as evident by calcaneal
    squeeze test or an os trigonum fracture. Both can be
    verified by triple phase nuclear bone scan.


KNEE INJURY


  • Soccer players can experience the gamut of overuse
    and acute knee problems. Youth players are prone to
    Osgood-Schlatter disease or any other patello-femoral
    tracking maladies seen in all running sports. Senior
    soccer players will suffer collateral ligament and
    meniscal injuries along with the pain of creeping
    osteoarthritis or patellar chondromalacia.
    •With the leg well grounded by the cleats of the soccer
    shoe, rapid rotational and flexion changes of the trunk
    or lower leg as in passing the soccer ball may produce
    sudden rotation of the femur relative to the fixed leg
    causing meniscal and/or knee ligament injuries. As
    mentioned earlier, tackling is a high-risk maneuver
    where knee ligament strain often occurs when a player
    is tackled with the loaded leg secured to the ground.

  • Soccer causes a higher incidence of meniscus injuries,
    but the extreme morbidity caused by ACL injuries has
    generated broad investigational research interest.

  • Soccer players rupture their ACL through direct con-
    tact or noncontact. The latter mechanism is the pre-
    dominant means for ACL injury in most sports other
    than skiing; however, Arendt and Dick (Arendt and
    Dick, 1995) reported that male collegiate soccer play-
    ers have an equal rate of contact versus noncontact
    ACL injuries. Female collegiate soccer players follow
    the usual trend for noncontact ACL injuries but as pre-
    viously mentioned dominates the males in this injury
    category.

  • The mechanism of noncontact ACL injury follows the
    common pattern of deceleration coupled with pivoting
    with internal or external rotation or the awkward land-
    ing of a varus or valgus collapsed knee. (Boden et al
    2000) studied the mechanism of ACL injury as they
    investigated 85 athletes with ACL tears. They reported
    72% of the ACL tears involved noncontact deceleration
    occurring at an average knee flexion of 23oat foot strike.
    (Delfico and Garrett 1998) write that at this angle of
    knee flexion the maximal anterior tibial translation


528 SECTION 6 • SPORTS-SPECIFIC CONSIDERATIONS

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