CHAPTER 89 • SOCCER 529
occurs coupled with maximal eccentric quadriceps con-
traction to supply the force in the noncontact rupture of
the ACL.
- Contact ACL injuries in male soccer players follow a
pattern of collision with a valgus stress to the knee
resulting in associated injuries to the menisci, the col-
lateral ligaments, and the articular cartilage. - In the cases of chondral injuries, extreme stress is
placed on the articular cartilage through repetitive
abrasive wear of high velocity pivoting and decelera-
tion or the acute disruption of the deep cartilage ultra-
structure by large shear forces. Chondral lesions are
more often seen on the femoral condyle. Acute X-ray
imaging for a swollen knee should include tunnel
views to rule out an osteochondral fracture and if pres-
ent, should be staged by knee MRI and co-managed
with an orthopedic surgeon.
LOWER LEG INJURY
•A soccer player’s lower leg is vulnerable to abrasions,
contusions, and fractures. Shin guards have become
the only mandatory protective devices in soccer, but
serve primarily to protect the leg from minor soft
tissue injuries. (Boden et al) reported in his series of
soccer tibial and fibular fractures that 90% occurred in
athletes wearing shin guards.
- The more typical scenarios involving lower leg
injuries of any severity occur with aggressive slide
tackles from behind with injury to the offensive
player’s fixed leg or when opposing players contest a
loose ball in scoring position where a haphazard
swinging kick or a lunging foot results in high veloc-
ity contact (Boden, 1998).
•With combined tibia and fibula fractures the soccer
player is sidelined on average of 40 weeks. Isolated
fibula and tibia fractures return to competitive play on
average of 18 and 35 weeks, respectively. Both com-
bined tib-fib fractures and isolated tibial fractures
have a high incidence of recovery complications
(Boden, 1998).
•Overuse injuries to the lower extremity span the broad
differential for exertional lower leg pain. These
include but are not limited to compartment syndrome,
medial tibial stress syndrome (MTSS), and stress frac-
tures. - Anterior tibial compartment syndrome can be insidi-
ous with the effort dependent running pain that
reduces performance over several months to years. Or
in another scenario, an acute compartment syndrome
may occur when a player sustains a high velocity kick
to the protected or unprotected anterior or lateral
lower leg. In both cases, the player may describe
lower leg pain with tingling and/or weakness extend-
ing on to the dorsum of the foot. Diagnostic stryker
compartment testing and comanagement with an
orthopedic surgeon should follow.
- Normal connective tissue and bone adaptation occurs
in cyclic progression. Injury patterns for MTSS and
stress fractures are multifactorial and are exercise
dose dependent where extremes of frequency, inten-
sity, and duration are common contributors. - Stress fractures and MTSS are diagnostic challenges
best differentiated by triple phase bone scan and
treated with varying degrees of activity modification.
GROIN INJURY
- The mechanism of groin injury in the soccer athlete is
associated with the ball manipulation skills where the
leg gets overstretched at the groin while the hip is
abducted and externally rotated, sometimes against an
opposing force, such as the ground or the opponent.
This process of overstretching compromises the ado-
lescent’s apophysial pelvic ring or pubic attachments
or in the case of the senior player the muscular—
tendonous attachments. - Groin pain can follow a pattern of overuse that starts
with adductor muscle tendonopathy. These cases
should be distinguished from osteitis pubis and sports
hernias, which can present with a similar pain pattern.
•Hip flexor strain to the iliopsoas is common in
soccer and is characterized by deep groin pain with
an occasional snapping hip sensation or pain exten-
sion onto the anterior thigh. Treatment consists of
relative rest with stretching of the hip flexors and
rotators. An iliopsoas strengthening program should
precede the return to competitive play (Morelli and
Smith, 2001). - Groin disruption was named Gilmore’s Groin in 1980
following the successful treatment of three profes-
sional soccer players who had been sidelined with pain
for 3 months. Clinical symptoms include the insidious
unilateral pain in the adductor region that progresses
with activity and follows a course of post activity
aggravation getting out of bed or the car. Examination
findings are minimal and variable but may include ten-
derness and dilation of the internal inguinal ring on
scrotal hernia palpation. The features of this condition
include torn external oblique aponeurosis, torn con-
joined tendon—conjoined tendon tear from the pubic
tubercle, dehiscence between conjoined tendon and
inguinal ligament, and no palpable hernia. Diagnosis
should include stork radiographs to evaluate pelvic sta-
bility. Cases that fail the rehabilitative process should
go to surgery for repair (Gilmore, 1998).