phospholipids matrix is an important factor in apoptosis; along with homeostatic
mechanism responsible for preserving membrane lipid composition and asymmetry.
Early apoptotic events are associated with disturbances in the lipid bilayer matrix
generating changes in membrane permeability of mitochondrial membrane in
particular.
sPleen recYcles BoDY
The body recycles the cellular materials from cell death by the process of
autolysis, also called self-digestion. It is kind of obvious what happens to the
macrophages that “eat” the body during a Die-off and how they recycle resources
to the body. Calculation of the influx of monocytes into the spleen and of the local
production of macrophages showed that under steady-state conditions, 55% of the
spleen macrophage population is supplied by monocyte influx and 45% by local
production. This means that there is a dual origin of spleen macrophages. The
mean turnover time calculated with the value for the efflux of spleen macrophages
is 6.0 days. Since worn out red blood cells are ingested by phagocytic cells in the
liver and spleen, I suspect that macrophages are also absorbed back into the system
in this way. Perhaps they too undergo apoptosis and are taken up by phagocytic
cells in the liver and spleen. Both the liver and spleen are capable of generating
ecstasy, quite possibly when there is a fresh supply of extra ATP recycled from an
increase in the breakdown of cells.
apoptosis is a normal function of the immune system. It is apparent in
metamorphosis that the body cannibalizes itself by cell death through oxidation
leading to apoptosis and immune phagocytosis. Recycling the constituents of cells
is probably a far more energy efficient form of nutrition than the digestion of
food. And considering that the digestive system is disrupted with the hyper-SNS,
it makes sense that during metamorphosis the body transforms itself through self-
digestion. The macrophages eat inferior cells and scavenger enzymes breakdown
used and damaged proteins into their component parts for reuse by the cell. Since
the energy of the body has shifted toward self-digestion, this is the primary reason
why our diet during metamorphosis needs to be light and nutrient, rather than
complex and hard to digest. Digestion of food will take energy away from the self-
catabolic digestive process.
The most important point I can emphasize is to not presume the catabolic
breakdown of cells, and the increased free radical load during metamorphosis
as pathological, for this will only increase our stress level. We cannot regenerate
without going through the dissolution. Since metamorphosis is a natural process
which is “self-governing,” our job in working-with what is happening is to first not
increase the free radical burden on the body with harmful substances and secondly
to increase our antioxidant intake and distressing techniques. We all know the
most harmful free radical inducers by now, they are: smoking, pollution, alcohol,
charred meat, hydrogenated oils, aged cheeses, rancid fats, sugar and stress.