cancer, specifically to revive mitochondrial activity in tumor cells, which generate
their energy anaerobically via gluconeogenesis, producing lactic acid.
Also along the lines of mitochondrial reactivation for cancer treatment is
ongoing research at the University of Alberta on using Dichloroacetic acid (DCA)
to kill cancer cells, but not normal cells. Evangelos Michelakis and his colleagues
discovered that this simple cheap molecule, DCA, worked to reactivate the
apoptosis mechanism of cancer cells, causing tumors in rats to shrink 70% in three
weeks. Mitochondrial reactivation represents an entirely new exciting approach to
treating cancer.
When mitochondrial DNA is oxidized and cellular energy falls, the cells
vulnerability to excitotoxic neurotransmitters is increased. Oxidative stress can
also trigger inflammatory cyctokines which induce the production of nitric oxide.
Acetyl-L-carnitine and alpha lipoic acid (and DHLA) are believed to play a role
in slowing mitochondrial oxidation and apoptosis. In research, the activity and
energy level of old rats taking these same supplements almost doubled, and memory
and cognitive function improved. By providing more “mental energy,” studies
show that animals receiving supplements are much more readily able to learn new
things as well, even at an advanced age. N-acetylcysteine (NAC) supplementation
enhances glutathione levels and mitochondrial respiration. Mitochondrial energy
production is also supported by magnesium and vtaimins B1, B2, B3, A, C, E, K
and folic acid.
See Dr. Ward Dean’s articles on Mitochondrial Restoration in the Research Center
at http://www.vrp.com
Seven Dimensions of Fitness: A Guide to Anti-Aging, Exercise, and Supernutrition by
Chris Kilham
Cracking the Metabolic Code by James B. LaValle and Stacy Lundin Yale
http://www.acu-cell.com/—Great nutrition information