I.1. BACTERIA: PATHOGENICITY FACTORS – 67
Gilbert, P., J. Das and I. Foley (1997), “Biofilm susceptibility to antimicrobials”, Advances in
Dental Research, Vol. 11, No. 1, pp. 160-167, April.
Girish, K.S. et al. (2004), “Isolation and characterization of hyaluronidase a ‘spreading factor’
from Indian cobra (Naja naja) venom”, Biochimie, Vol. 86, No. 3, pp. 193-202, March.
Gladysheva, I.P. et al. (2003), “Coevolutionary patterns in plasminogen activation”, Proceedings
of the National Academy of Sciences USA, Vol. 100, No. 16, pp. 9 168-9 172,
http://www.pnas.org/content/100/16/9168.full.pdf.
Glandorf, D.C.M. et al. (2001), “Effect of genetically modified Pseudomonas putida WCS358r on
the fungal rhizosphere micro of field-grown wheat”, Applied and Environmental Microbiology,
Vol. 67, No. 8, pp. 3 371-3 378, http://dx.doi.org/10.1128/AEM.67.8.3371-3378.2001.
Glomski, I.J. et al. (2002), “The Listeria monocytogenes hemolysin has an acidic pH optimum to
compartmentalize activity and prevent damage to infected host cells”, Journal of Cell Biology,
Vol. 156, No. 6, pp. 1 029-1 038, March.
Goodwin, A.C. et al. (2008), “Expression of the Helicobacter pylori adhesin SabA is controlled
via phase variation and the ArsRS signal transduction system”, Microbiology, Vol. 154, Pt. 8,
pp. 2 231-2 240, http://dx.doi.org/10.1099/mic.0.2007/016055-0.
Goosney, D.L., R. DeVinney and B.B. Finlay (2001), “Recruitment of cytoskeletal and signalling
proteins to enteropathogenic and enterohemorrhagic Escherichia coli pedestals”, Infection and
Immunity, Vol. 69, No. 5, pp. 3 315-3 322, http://dx.doi.org/10.1128/IAI.69.5.3315-3322.2001.
Gouin, E. et al. (1999), “A comparative study of the actin-based motilities of the pathogenic
bacteria Listeria monocytogenes, Shigella flexneri and Rickettsia conorii”, Journal of Cell
Science, Vol. 112, Pt. 11, pp. 1 697-1 708, June.
Gradon, J.D., J.G. Timpone and S.M. Schnittman (1992), “Emergence of unusual opportunistic
pathogens in AIDS: A review”, Clinical Infectious Diseases, Vol. 15, No. 1, pp. 134-157, July.
Grange, P.A. et al. (2002), “Evaluation of receptor binding specificity of Escherichia coli K88
(F4) fimbrial adhesin variants using porcine serum transferrin and glycosphingolipids as model
receptors”, Infecton and Immunity, Vol. 70, No. 5, pp. 2 336-2 343, May.
Greenberg, E.P. (2003), “Bacterial communication and group behavior”, Journal of Clinical
Investigation, Vol. 112, No. 9, pp. 1 288-1 290, http://dx.doi.org/10.1172/JCI200320099.
Guhathakurta, B. et al. (1999), “Adhesion and invasion of a mutant Shigella flexneri to an
eukaryotic cell line in absence of the 220-kb virulence plasmid”, FEMS Microbiology Letters,
Vol. 181, No. 2, pp. 267-275, December.
Hackstadt, T. and J.C. Williams (1981), “Biochemical strategem for obligate parasitism of
eukaryotic cells by Coxiella burnetti”, Proceedings of the National Academy of Sciences USA,
Vol. 78, No. 5, pp. 3 240-3 244, May.
Hancock, L.E. and M.S. Gilmore (2002), “The capsular polysaccharide of Enterococcus faecalis
and its relationship to other polysaccharides in the cell wall”, Proceedings of the National
Academy of Sciences USA, Vol. 99, No. 3, pp. 1 574-1 579, February.
Harb, O.S. and Y. Abu Kwaik (1999), “Probing the microenvironment of intracellular bacterial
pathogens”, Microbes and Infection, Vol. 1, No. 6, pp. 445-453, May.
Hardy, G.G., S.M. Tudor and J.W. St. Geme III (2003), “The pathogenesis of disease due to
nontypeable Haemophilus influenzae”, Methods of Molecular Medicine, Vol. 71, pp. 1-28.
Harris, T.O. et al. (2003), “A novel streptococcal surface protease promotes virulence, resistance
to opsonophagocytosis, and cleavage of human fibrinogen”, Journal of Clinical Investigation,
Vol. 111, No. 1, pp. 61-70, January, http://dx.doi.org/10.1172/JCI200316270.