© Springer International Publishing AG 2017 261
T. Angelone et al. (eds.), Chromogranins: from Cell Biology to Physiology
and Biomedicine, UNIPA Springer Series, DOI 10.1007/978-3-319-58338-9
A
Acute myocardial infarction (AMI), 172, 181
Adrenergic drugs, 102
Adrenocorticotropic hormone (ACTH),
41, 215, 217
African American Study of Kidney Disease
and Hypertension (AASK) trial
cohort, 203
Angiogenesis
chromogranin A
proteases, 88
tumors, 9, 90–92
VEGF-and FGF-2, RAR assay,
87–88
secretoneurin, 30–31
Anti-adrenergic drug therapy, 102
Antibiotics, 50, 57–58
Antimicrobial peptides (AMPs)
CGA-derived peptides
antibiotics, 57–58
bacterial proteases, 56–57
biomaterials, 62–63
catestatin, 53, 55, 56
chromofungin, 53, 55–56
location, sequence and net charge,
53, 54
SIRS and acute circulatory failure
criteria, 59–61
VS-I, 53–56
innate and adaptive immunity, 5, 50, 51
Anti-Thy1.1 antibody, 92
Arfaptin 1, 42, 43
Arlequin, 197
Atrial natriuretic peptide (ANP), 104, 161
Autophagy, 172
B
Bacterial proteases, 56–57
Basic fibroblast growth factor (bFGF), 162
β-adrenergic receptors (β-ARs), 116
Bin/Amphiphysin/Rvs (BAR) domains, 42, 43
Bovine chromogranin A (bCgA) protein,
216, 217
Bovine fibroblast growth factor (bFGF2), 224
Brain natriuretic peptide (BNP), 104, 105,
161, 171C
Carboxypeptidase B, 88
Carboxypeptidase E (CPE), 42
Carcinoids, 89
Cardioprotection, CgA derived peptides
catestatin, 181–184
chromofungin, 184
ischemia/reperfusion injury
apoptosis, 172, 175
autophagy, 172
Ca2+ overload phenomenon, 174
complex biochemical and mechanical
mechanisms, 172
inflammatory process, 175
mPTP, 174
myocardial ischemia, 173
necrosis, 172, 175
NFκB, 175
nitric oxide, 173–175
no-reflow phenomenon, 175
oxidative stress, 173, 174
peroxynitrite, 174
ROS/RNS, 173, 175Index