[82] Goldberg DE, Slater AF, Cerami A, Henderson GB. Hemoglobin degradation in the
malaria parasite Plasmodium falciparum: an ordered process in a unique organelle.
Proceedings of the National Academy of Sciences of the United States of America.
1990; 87 (8):2931‐2935. doi:10.1073/pnas.87.8.2931
[83] Goldberg DE, Slater AF, Beavis R, Chait B, Cerami A, Henderson GB. Hemoglobin
degradation in the human malaria pathogen Plasmodium falciparum: a catabolic path‐
way initiated by a specific aspartic protease. The Journal of Experimental Medicine.
1991; 173 (4):961‐969. doi:10.1084/jem.173.4.961
[84] Dickerson RE, Geis I. Hemoglobin: Structure, Function, Evolution, and Pathology.
Menlo Park, CA: The Benjamin/Cummings Publishing Company; 1983. 176 p.
[85] Stamatoyannopoulos G, Nienhuis AW, Leder P, Majerus PW, editors. The Molecular
Basis of Blood Diseases. Philadelphia: W.B. Saunders Company; 1987. 747 p.
[86] Chugh M, Sundararaman V, Kumar S, Reddy VS, Siddiqui WA, Stuart KD, et al.
Protein complex directs hemoglobin‐to‐hemozoin formation in Plasmodium falciparum.
Proceedings of the National Academy of Sciences of the United States of America.
2013; 110 (14):5392‐5397. doi:10.1073/pnas.1218412110
[87] Sherman IW, Tanigoshi L. Incorporation of 14C‐amino‐acids by malaria (Plasmodium
lophurae) IV. In vivo utilization of host cell haemoglobin. International Journal of
Biochemistry. 1970; 1 (5):635‐637. doi:10.1016/0020‐711x(70)90033‐9
[88] Sherman IW. Amino acid metabolism and protein synthesis in malarial parasites.
Bulletin of the World Health Organization. 1977; 55 (2‐3):265‐276.
[89] Zarchin S, Krugliak M, Ginsburg H. Digestion of the host erythrocyte by malaria parasites
is the primary target for quinoline‐containing antimalarials. Biochemical Pharmacology.
1986; 35 (14):2435‐2442. doi:10.1016/0006‐2952(86)90473‐9
[90] Wernsdorfer WH, McGregor I, editors. Malaria: Principles and Practice of Malatiology,
Volume 1. Edinburgh: Churchill Livingstone; 1988. 1818 p.
[91] Naughton JA, Nasizadeh S, Bell A. Downstream effects of haemoglobinase inhibition in
Plasmodium falciparum‐infected erythrocytes. Molecular and Biochemical Parasitology.
2010; 173 (2):81‐87. doi:10.1016/j.molbiopara.2010.05.007
[92] Ting IP, Sherman IW. Carbon dioxide fixation in malaria—I. Kinetic studies in plas‐
modium lophurae. Comparative biochemistry and physiology. 1966; 19 (4):855‐869.
doi:10.1016/0010‐406x(66)90441‐5
[93] Polet H, Conrad ME. Malaria: extracellular amino acid requirements for in vitro growth
of erythrocytic forms of Plasmodium knowlesi. Proceedings of the Society for Experimental
Biology and Medicine Society for Experimental Biology and Medicine. 1968; 127 (1):251‐
- doi:10.3181/00379727‐127‐32666
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