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For Homeowners!received an application of a vaccine on
the tonsils and rectum. Afer the initial
vaccination, four further vaccine repeti-
tions were given to these fve deer over
an 11-month period.
To follow all the details of the vacci-
nation process and the subsequent tissue
and liquid analyses, you would need to
be an expert in antibody studies, im-
munohistochemistry, and other medical
disciplines. Given that, here is a brief,
condensed summary of the results.
In short, the control deer had a medi-
an survival of 602 days, while the vacci-
nated deer had a median survival of 909
days. So, now what does this all mean?
Do vaccinations work, and if so, how do
we do it?
Tis study shows that the approach
they used worked, at least partially. Te
inoculation was oral, and CWD trans-
mits via oral liquids (mucous). So that
part is a go. Another positive was the fact
that even though all the CWD-exposed
deer were housed together, groomed
each other, etc., one vaccinated deer did
not get CWD and is still alive. Another
plus was the fact that researchers used a
very high dose of CWD inoculum. Tey
wanted to make sure that all the deer had
CWD, so they used a dose much higher
than what a deer would be exposed to
in the wild. Tis led the researchers to
state that, “It is possible to speculate
that our current vaccination procedure
could have a greater degree of protection
against a CWD inoculum that refects a
dose more likely to occur in the wild.”
In other words, the vaccine they used
might work even better in the wild than
in the lab.
Te researchers also noted that in fu-
ture studies, they feel they can deliver the
inoculum in such a way as to get higher
antibody levels than those obtained in
this study. Higher antibody levels means
higher resistance to CWD.
Te big question is, how do you get
the vaccine into deer in the wild? Here
is what the researchers believe. “Subse-
quently, we found that the vaccine could
be administered mixed with food, so
deer can be dosed repeatedly without
anesthesia. Thus, it would be possible
to dose individual deer in a free-range
herd repeatedly, using food pellets con-
taining vaccine Salmonella lef in the
environment at appropriate times, given
the known tendency of herds to return
to the same foraging grounds at regularintervals.” Tis is an interesting thought,
because we know that baiting brings
deer together and enhances the transfer
of CWD via saliva. However, now we
hear that baiting with an appropriate
pellet with the vaccine (or perhaps corn,
my words here) might do the opposite
and be a way to vaccinate deer. I never
thought I’d see the day when baiting for
deer might be a good thing.
One last thought. Could a vaccinated
doe pass her anti-CWD antibodies on to
her unborn fawns? My thought is, prob-
ably. If so, then protection from CWD
would not involve continual inoculation
via food of all deer. In fact, one might
be able to slow the spread of CWD with
treatments that reach one-quarter of all
deer, or maybe much less than that.
What this initial study does is stimu-
late more research to refne just how this
can be done. Will that research happen?
Of course, because the answers found
here could also be answers for cures for
the prion diseases that humans get. In-
deed, as I said above, this frst step is ex-
tremely exciting. ❮❮❮For further information on the issues discussed,
go to http://www.knowhunting.comWWW.BOWHUNTER.COM 33