ENVIRONMENTAL EMERGENCIES
■ Adjuncts:
■ Place ice packs to groin, axillae, and neck.
■ Cooling blanket
■ If refractory, consider invasive methods.
■ Cardiopulmonary bypass
■ Gastric, pleural, or bladder lavage
■ IV benzodiazepines (or paralysis) to prevent shivering (which generates
heat); treat seizures
■ Intravenous fluids to CVP of 12 mL H 2 O or urine output = 0.5 mL/
kg/hr
■ Fluid requirements vary with underlying cause.
■ Dopamine for persistent hypotension despite adequate fluid resuscitation
■ Dantrolene: If suspected malignant hyperthermia.
■ Avoid
■ Antipyretics—noteffective and may be harmful!
■ Prophylactic steroids—noteffective.
■ Norepinephrine—causes vasoconstriction and decreases cutaneous
heat exchange.
■ Poor prognostic indicators:
■ Coma
■ AST > 1000 IU/L.
■ Delay in rapid cooling (morbidity and mortality are directly related to
the duration of hyperpyrexia)
■ DIC
■ Hypotension
■ Renal failure in <48 hours
■ Lactic acidosis
COMPLICATIONS
■ ARDS, electrolyte abnormalities, renal failure, rhabdomyolysis, hepatic
injury, DIC
■ If left untreated cerebral edema and multisystem organ failure will
develop.
A homeless man presents to your ED after walking over 3 miles in freezing
snow. He is complaining of numbness and swelling to his lower extremities.
On exam you see diffuse erythema, edema, and both clear and hemorrhagic
blisters. How will you treat this man’s blisters?
With frostbite, damaged tissue releases arachidonic acid breakdown products
(prostaglandins and thromboxane). These released products promote platelet
aggregation, leading to thrombosis and ischemia. Blisters should be left intact
or sterilely aspirated. Debride blisters only if broken.
COLD-RELATED INJURIES
There are four physiologic mechanisms of heat loss/gain (see Table 13.7).
Radiation is responsible for the majority of normalheat loss.
Antipyretics are noteffective
(and may be harmful) in
treatment of heat stroke!