RENAL AND GENITOURINARY
EMERGENCIES
■ Peripheral edema
■ Proteinuria—3+ or 4+ on dipstick
■ Fatty casts or oval fat bodies on UA: Related to associated hyperlipi-
demia
■ Hypoproteinemia
■ BUN and creatinine are often normal.
TREATMENT
■ May include fluid restriction, IV diuretics
COMPLICATIONS
■ Increased risk of thrombosis
■ The most notable is renal vein thrombosis, characterized by hema-
turia, flank pain, and worsening renal function.
HEMATURIA
Hematuria can be microscopic or gross in nature. Microscopic hematuria is
defined as >3 RBC/hpf on spun urine sediment, while gross hematuria corre-
lates to 1 mL of blood in 1 L of urine. Gross hematuria has a higher inci-
dence of serious underlying pathology.
Hematuria can be thought of as belonging to four main groups:
■ Traumatic
■ Hematologic: eg, coagulopathy, sickle cell disease
■ Renal: eg, glomerulonephritis, AVM
■ Postrenal: eg, stones, bladder carcinoma, BPH, UTI
Table 18.1 lists the most common etiologies of hematuria by age group.
SYMPTOMS/EXAM
■ Vary with underlying etiology
Renal vein thrombosis should
be considered in all patients
with nephrotic syndrome
presenting with hematuria,
flank pain, and worsening
renal function.
TABLE 18.1. Common Causes of Hematuria by Age
Common Causes of Hematuria by Age
<20 yr Glomerulonephritis
UTI
20–40 yr GU trauma
Malignancy
Stone
UTI
40–60 yr Carcinoma (bladder, kidney)
Stone
UTI
BPH (males)