Ultrasound and CT scans.
Hematocritand hemoglobin levels to monitor for bleeding.
Peritoneal fluid may contain increased levels of pancreatic enzymes.
The stools are often bulky, pale, and foul-smelling. Fat content of stools varies
between 50% and 90% in pancreatic disease; normally, the fat content is 20%.
Medical Management
Management of the patient with acute pancreatitis is directed toward relieving
symptoms and preventing or treating complications. All oral intake is withheld
to inhibit pancreatic stimulation and secretion of pancreatic enzymes.
Parenteral nutrition is usually an important part of therapy, Nasogastric
suction may be used to relieve nausea and vomiting, to decrease painful
abdominal distention and paralytic ileus, and to remove hydrochloric acid so
that it does not enter the duodenum and stimulate the pancreas.
Histamine-2 (H2) antagonists to decrease pancreatic activity by inhibiting HCl
secretion.
Pain Management
Adequate pain medication is essential during the course of acute pancreatitis
to provide sufficient pain relief and minimize restlessness, which may
stimulate pancreatic secretion further. Morphine and morphine derivatives are
often avoided because it has been thought that they cause spasm of the
sphincter of Oddi; meperidine (Demerol) is often prescribed because it is less
likely to cause spasm of the sphincter
Intensive care
Correction of fluid and blood loss and low albumin levels is necessary to
maintain fluid volume and prevent renal failure. The patient is usually acutely
ill and is monitored in the intensive care unit, where hemodynamic monitoring
and arterial blood gas monitoring are initiated. Antibiotic agents may be
prescribed if infection is present; insulin may be required if significant
hyperglycemia occurs.
Respiratory Care
Aggressive respiratory care is indicated because of the high risk for elevation
of the diaphragm, pulmonary infiltrates and effusion, and atelectasis.
Hypoxemia occurs in a significant number of patients with acute pancreatitis