Mood Disorders and Suicide 203
However, genes are not destiny. The environment clearly plays an important
role in whether a person will develop depression (Eley et al., 1998; Hasler et al.,
2004; Rice, Harold, & Thaper, 2002; Wender et al., 1986). Even with identical
twins, if one twin is depressed, this does not guarantee that the co-twin will also be
depressed—in spite of their having basically the same genes. Whether a person gets
depressed depends partly on his or her life experiences, including the presence of
hardships and the extent of social support.
The environment plays a key role not only in whether the genes contribute to
depression, but also in how the genes have their effects. In some cases, genetic fac-
tors may affect depression indirectly—by disrupting specifi c aspects of normal func-
tioning that in turn trigger the disorder. For example, researchers have found that
genetics may infl uence whether a person has disrupted sleep (Hasler et al., 2004;
Modell et al., 2003), and adolescents who went on to develop the disorder (investi-
gated in longitudinal studies) did have disrupted sleep.
Psychological Factors
Particular ways that people think about themselves and events, in concert with
stressful or negative life experiences, can increase the risk of depression. In the fol-
lowing sections we consider psychological factors that can infl uence whether a per-
son develops depression; these factors range from biases in attention to the effects
of different ways of thinking to the results of learning.
Attentional Biases
Some people see a glass that is half full of water as being half empty. Similarly, some
people focus their attention—consciously or unconsciously—on stimuli that are sad.
People who are depressed are more likely to pay attention to sad and angry faces
than to faces that display positive emotions (Gotlib, Kasch, et al., 2004; Gotlib,
Krasnoperova, et al., 2004; Leyman et al., 2007); people who do not have a psy-
chological disorder spend equal time looking at faces that express different emo-
tions. This attentional bias has also been found for negative words and scenes, as
well as for remembering depression-related—versus neutral—stimuli (Caseras et al.,
2007; Gotlib, Kasch, et al., 2004; Matt, Vasquez, & Campbell, 1992; Mogg, Brad-
ley, & Williams, 1995). Such an attentional bias may leave depressed people more
sensitive to other people’s sad moods and to negative feedback from others (or
even a lack of positive feedback, as occurs when a person fails to smile when
greeting you), compounding their depressive thoughts and feelings.
Dysfunctional Thoughts
As discussed in Chapter 2, Aaron Beck proposed that cognitive distortions are the
root cause of many disorders. Beck (1967) has suggested that people with depres-
sion tend to have overly negative views about (1) the world, (2) the self, and (3) the
future, referred to as the negative triad of depression. These distorted views can
cause and maintain chronically depressed feelings and depression-related behaviors.
For instance, a man who doesn’t get the big raise he hoped for might respond with
cognitive distortions that give rise to dysfunctional thoughts. He might think that
he isn’t “successful” because he didn’t get the raise, and therefore that he must be
worthless (notice the circular reasoning). This man is more likely to become de-
pressed than he would be if he didn’t have such dysfunctional thoughts.
Rumination
While experiencing negative emotions, some people refl ect on these emotions; dur-
ing such ruminations, they might say to themselves: “Why do bad things always
happen to me?” or “Why did they say those hurtful things about me—is it something
I did?” or “Should I have spoken more during the discussion?” (Nolen-Hoeksema &
Morrow, 1991). Such ruminative thinking has been linked to depression (Just &
Alloy, 1997; Nolen-Hoeksema, 2000; Nolen-Hoeksema & Morrow, 1991, 1993).
Studies have examined the relationship between depression and specifi c forms of
rumination, such as stress-reactive rumination, which is ruminating about negative
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