HETEROSEXUAL
naturally normal range of gender variationIf insufficient androgens (male hormones) are circulated during fetal
development, chances increase that a genetically male fetus will develop a
feminized brain that later dictates character traits describing effeminate,
homosexual or transsexual males respectively according to decreasing levels
of hormone. To the contrary, if the fetus is female the brain can be exposed
to excessive androgens that cause masculinized brain structures favoring
male behavior and/or thought patterns, so that the child may become ‘Tom-
boyish’, feminine-lesbian, masculine-lesbian, or even transsexual on
maturation.v In addition, there is second surge of masculinizing hormones
permanently affecting the brain at 6 months of gestation if the fetus is a boy,
which can then result in hypermasculine homosexuality such as Rock Hudson
or Raymond Burr.
The chart is an accurate oversimplification of the leading hypothetical
construct as to cause and fairly represents an extremely complicated unified
enmeshment of data that supports the thesis — albeit, it does not adequately
stand alone as to causality and effect due to components of genetic,
immunological and environmental influences yet to be completely defined.
Male Fetal Brain
Proclivity- Hypermasculine
- Masculine
- Effeminate
- ---------------
- Bisexual
- Homosexual
- Transsexual
Fetal Androgen Level
(6-8 wks)- More Than Sufficient for male , extreme
for female - Sufficient for male , too high for female
- Adequate for male , excessive for female
- ----------------
- Insufficient for male, too much for
female - Deficient for male, normal for female
- Absent or too little
Female Fetal Brain
Proclivity- Transsexual
- Lesbian
- Bisexual
- ----------------
- Masculine traits
- Feminine
- Hyperfeminine